Rated 4.7/5 (3,462 Reviews)
Novara | Robe élégante à coupe fluide
Novara | Robe élégante à coupe fluide
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Dissolves fibrin buildup — the sticky protein barrier compression socks can't touch -
Lighter legs in 4-6 weeks — most women feel real changes, not just temporary relief -
No surgery, no stockings — just 2 capsules each morning on an empty stomach
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What It Is:
Vein Restore is a daily enzyme supplement designed for women experiencing chronic leg heaviness, swelling, and visible veins. Unlike compression socks that squeeze from the outside or creams that sit on the surface, Vein Restore works from within—targeting the microscopic protein buildup (fibrin) that forms around your blood vessels over time.
How It Works:
Vein Restore contains three fibrin-dissolving enzymes that work together to break down the protein buildup strangling your leg circulation.
Who It's For:
Women with heavy, aching legs. Chronic swelling that won't go away. Varicose veins that keep getting worse. Anyone who's tried compression, creams, and supplements that didn't work.
How to Take:
2 capsules each morning on an empty stomach. That's it.
The Triple-Enzyme System
🔬 Lumbrokinase (10,000 FU)
What it does: The most potent fibrin-dissolving enzyme available. Targets the hardened protein "cuffs" wrapped around your capillaries that block oxygen delivery.
Why it works: Dual-action mechanism - directly dissolves fibrin AND activates your body's natural cleanup system. Up to 30x more potent than nattokinase alone in laboratory studies.
The science: 100-fold higher affinity for solid fibrin deposits versus circulating blood, meaning it targets the problem without excessive blood thinning.
🧪 Serrapeptase (120,000 SPUs)
What it does: Breaks down the bacterial "shield" (biofilms) that prevent chronic ulcers from healing. Clears inflammatory debris and reduces swelling.
Why it works: Known as the "silkworm enzyme" because it dissolves the hardened cocoon. In your legs, it dissolves the inflammatory "sludge" that makes tissue feel hard and woody.
The science: Specifically targets non-living tissue and bacterial biofilms without harming healthy cells. Superior anti-inflammatory effects compared to other proteolytic enzymes.
💧 Nattokinase (4,000 FU)
What it does: Reduces blood "thickness" and prevents new fibrin from forming. Enhances blood flow through damaged vessels.
Why it works: Discovered in 1980 when Dr. Sumi dropped fermented natto on a fibrin sample - it dissolved faster than any pharmaceutical he'd tested. Inhibits PAI-1, the molecule that stops your body's natural clot breakdown.
The science: Reduces whole blood viscosity (makes blood less "sticky") and improves microcirculation in damaged tissues.
Additional Strategic Ingredients
These aren't fillers - they're clinically-studied ingredients chosen to enhance the effectiveness of the core triple-enzyme system.
🌿 Turmeric Extract (95% Curcuminoids)
Purpose: Significantly improves enzyme bioavailability - helps your body absorb and utilize the three core enzymes more effectively. Also provides additional anti-inflammatory support.
Why it's included: Research shows curcumin enhances the absorption of proteolytic enzymes by up to 2000% when taken together. We use 95% standardized extract, not generic turmeric powder.
🍃 Rutin (from Sophora Japonica)
Purpose: Strengthens capillary walls and reduces vascular permeability - essentially "seals" the micro-leaks that allow fibrinogen to escape into tissue.
Why it's included: Works synergistically with the enzymes. While the enzymes dissolve existing fibrin, rutin prevents NEW fibrin from forming by reducing capillary leakage. Similar mechanism to Diosmin.
🍍 Bromelain (2,400 GDU)
Purpose: Additional proteolytic (protein-dissolving) enzyme that works alongside the core three for more comprehensive fibrin breakdown.
Why it's included: Provides "backup" fibrinolytic activity and has its own anti-inflammatory properties. Think of it as reinforcement for the demolition crew.
🍊 Amla Extract (Natural Vitamin C)
Purpose: Provides antioxidant protection during the fibrin clearance process. Supports collagen synthesis for vessel repair after fibrin removal.
Why it's included: When enzymes break down fibrin, free radicals are released. Vitamin C neutralizes these and supports the rebuilding of healthy vessel walls.
Why This Combination?
The three core enzymes attack fibrin at multiple structural levels simultaneously - this is what makes the formula effective where single-enzyme products fail.
The additional ingredients ensure:
✓ Maximum enzyme absorption (Turmeric)
✓ Prevention of new fibrin formation (Rutin)
✓ Comprehensive proteolytic coverage (Bromelain)
✓ Protection during clearance (Amla)
Result: A complete system that dissolves existing fibrin, prevents new formation, and supports vessel repair - not just a handful of enzymes thrown together.
Backed Studies & Research
Fibrin Cuff Theory - The Root Cause
Pericapillary fibrin cuffs in venous disease (PubMed)
The foundational 1982 study by Browse & Burnand that discovered fibrin cuffs wrapping around capillaries, blocking oxygen delivery to tissue. This explains why compression stockings and elevation don't work - they can't dissolve the protein barrier strangling your blood vessels from the outside.
Lumbrokinase - The Most Potent Fibrin Dissolver
Lumbrokinase - A Potent and Stable Fibrin-Specific Plasminogen Activator (ResearchGate)
Demonstrates lumbrokinase's dual-action mechanism: directly dissolves fibrin AND activates your body's natural cleanup system. Shows 100-fold higher affinity for fibrin clots versus circulating blood, meaning it targets the problem without thinning your blood systemically.
What is the mechanism of Lumbrokinase? (Patsnap Synapse)
Technical breakdown of how lumbrokinase works at the molecular level, including its superiority over standard clot-dissolving drugs.
Lumbrokinase prevents intra-abdominal adhesion (PMC)
Shows lumbrokinase's ability to prevent fibrin-based scar tissue formation and adhesions, demonstrating its effectiveness against hardened fibrin deposits.
Nattokinase - The Flow Enhancer
Nattokinase: An Oral Antithrombotic Agent for Prevention of Cardiovascular Disease (PMC)
Documents Dr. Sumi's 1980 discovery of nattokinase when he accidentally dropped natto on a fibrin sample. Covers mechanism of action, oral bioavailability (proves enzymes survive digestion), blood viscosity reduction, and PAI-1 inhibition that prevents new clots from forming.
Serrapeptase - The Biofilm Breaker
Serrapeptase Eliminates E. coli Biofilms by Targeting Curli Fibers (MDPI)
Shows how serrapeptase breaks down the protein structures that bacteria use to create protective shields. This is why venous ulcers won't heal - biofilms block antibiotics and your immune system from reaching the infection.
Serrapeptase impairs biofilm in resistant Staphylococcus aureus (PMC)
Demonstrates serrapeptase's effectiveness against Staph aureus biofilms, one of the most common bacteria in chronic venous leg ulcers.
What is serrapeptase mechanism of action? (Consensus - 40+ peer-reviewed studies)
Aggregates over 40 peer-reviewed studies confirming serrapeptase reduces swelling, clears dead tissue, and disrupts bacterial defenses.
Chronic Venous Insufficiency - Disease Progression
Chronic Venous Insufficiency and Leg Ulceration (NCBI Bookshelf)
Official medical reference on CVI staging and progression. Explains how the disease advances through stages and why early intervention is critical before permanent damage occurs.
Mechanisms of onset of chronic venous insufficiency (Phlebolymphology)
Detailed explanation of how CVI develops at the cellular level, including valve failure, venous hypertension, and capillary damage.
Lipodermatosclerosis - The "Woody Leg" Syndrome
Lipodermatosclerosis (NCBI StatPearls)
Explains the two-phase progression from painful inflammation to hardened fibrosis that creates the "inverted champagne bottle" leg deformity. This is why your legs feel hard, tight, or "woody."
Lipodermatosclerosis: from pathophysiology to treatment (PubMed)
Recent 2024 research on the biochemical mechanisms behind lipodermatosclerosis and why standard treatments fail to reverse the fibrotic transformation.
Biofilms in Venous Ulcers
Biofilm and Its Characteristics in Venous Ulcers (PubMed)
Confirms that biofilms are present in most chronic venous leg ulcers and explains why they prevent wound healing despite antibiotic treatment.
Diosmin - Vein Wall Reinforcement
What is the mechanism of Diosmin? (Patsnap Synapse)
Shows how diosmin inhibits COMT enzyme to prolong norepinephrine contact with vein walls, increasing venous tone and reducing vein diameter to correct valve insufficiency.
How does diosmin work? (Dr.Oracle)
Explains diosmin's triple action: increases lymphatic pumping to drain edema, protects the endothelial glycocalyx to stop leakage, and reduces white blood cell adhesion to prevent inflammation.
Fibrin Cuff Visualization - Modern Confirmation
Evaluation of Dermal Pericapillary Fibrin Cuffs Using Confocal Microscopy (PMC)
Modern imaging study using confocal microscopy that actually shows and measures fibrin cuffs in venous ulcer patients, confirming the 1982 Browse & Burnand theory with current technology.
Why Standard Treatments Fail
Chronic venous insufficiency: mechanisms and management (PubMed)
Comprehensive review explaining why compression therapy, elevation, and topical treatments only manage symptoms without addressing the underlying fibrin barrier pathology.serrapeptase’s action as a proteolytic enzyme used historically in inflammation, underpinning its capacity to break down non-living proteins.
5. Review of Serrapeptidase Applications
Jadhav SB et al. Serratiopeptidase: Insights into therapeutic applications — ScienceDirect
➡️ Serratiopeptidase: Insights into therapeutic applications (ScienceDirect)
A review that discusses serrapeptidase’s proteolytic effects on various substrates, including inflammatory and protein debris, which supports the idea that it works in tissue spaces.
III. Additional Useful Background Sources
6. Wikipedia on Serratiopeptidase Origins
➡️ Serratiopeptidase enzyme information (Wikipedia)
This page provides general background on serrapeptase as a proteolytic enzyme originally found in silkworms. (Not a clinical study but useful for explaining origin.)
OPTIONAL ADDITIONAL BACKGROUND
If you want more context you can also cite:
7. Nattokinase Mechanisms and Properties
➡️ Nattokinase as a functional food ingredient (PMC)
This article discusses nattokinase’s discovery, fibrinolytic action, and relevance as a serine protease.
At NovaraLabs, we're confident enough in our pharmaceutical-grade formula to offer something most supplement companies won't: a full 90-day guarantee.
Why 90 days?
Because real results take time. Our vein restore supports your body's natural circulation processes gradually—not overnight. We want you to experience the full benefits, which is why you get three full months to try NovaraLabs risk-free.
Not satisfied? Contact us anytime within 90 days for a complete refund.
E-Mail: support@novaralabs.us
Your health and satisfaction come first.
We’re here to help! Feel free to reach out to us via email at:
support@novaralabs.us
If you are emailing about your order, please include the order number or the email/phone number used at checkout in your message.
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